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Ikossi, Stanford Vascular SurgeryO  =&7Brachiocephalic Vessel Reconstruction: Chapter 91, Rutherford s Vascular Surgery*S&&(* D@Danagra Georgia Ikossi, MD Stanford Surgery Resident, R3 1/30/06A&: BackgroundAortography: 1/3 of pts have significant lesions of the vessels of arms or head Small fraction of these are SAT (supra-aortic trunks) lesions Critical territory: cerebral hemispheres, ocular, vertebrobasilar, upper extremity and cardiac vascular beds. Z$ /Anatomy $Diseases of the Supra-Aortic Trunks%%(Atherosclerotic disease Inflammation Takayasu s Arteritis Radiation Dissection Aneurysm Thoracic outlet syndrome (SCA only) Congenital lesionsB%ZZKZ%K 3Atherosclerotic Disease of the Supra-Aortic Trunks44(50-61yo mean age (younger than other occlusive atherosclerotic lesions) Smoking Hypertension Diabetes CAD concomitant Radiation !3Atherosclerotic Disease of the Supra-Aortic Trunks44(Severe disease 75% stenosis ulcerated plaque thrombus in lumen Multivessel disease: may be  spillover from aortic arch disease into vessel ostia Single vessel disease: most commonly left subclavian BZ0ZZ0,@ B; Symptoms: SAT occlusive disease!!(Multivessel disease vertebrobasilar ischemia from low flow Single vessel disease upper extremity emboli Proximal subclavian disease subclavian-vertebral steal Innominate disease steal from carotid to subclavian@>$  " SAT Aneurysms:Complications: thrombosis, embolus, compression of surrounding structures, rupture Traditionally syphilitic in origin (rare now) Innominate and common carotid aneurysms or dissections Extension of dilation of proximal ascending aorta or thoracoabdominal aorta Takayasu s Arteritis @PaPPa# SAT AneurysmsSubclavian Artery Aneurysms 2% of all peripheral aneurysms Middle or distal 1/3 of vessel Extrathoracic: post-stenotic dilation, TOS Intrathoracic: atherosclerotic, cystic medial necrosis, Marfan s, infection (TB, syphillis)\PPPP]  + )Traumatic AneurysmsFalse aneurysms Short segment dissections Secondary to deceleration injury Mid-subclavian pseudoaneurysms from penetrating iatrogenic injuryZ* Aneurysms Kommerell s diverticulum: diverticulum at origin of anomalous artery off the arch (usually RSA) Takayasu s Arteritis: 3 vessel disease, proximal, inflammation, initially occlusive, later aneurysmal.:IS>    DiagnosisPhysical examination: Bilateral brachial cuff pressures Auscultation for bruits Palpate pulses carotid, superficial temporal and upper extremity Duplex of arch (screening tool) Digital Subtraction Angiography MRI, CT AngioBZ|ZPZ|P DiagnosisRoutine preoperative studies may include: TEE (evaluate aorta, r/o embolic source) CT of head (r/o recent infarct) Coronary angiogram (r/o CAD)&*f*f>?% Indications for Reconstruction(pSymptomatic TIAs: occular, hemispheric or vertebrobasilar (emboli or low-flow) Upper extremity ischemia Limb threateneing ischemia (occlusive disease or emboli) Steal syndrome L SCA occlusion: vertebral L SCA Innominate occlusion: R CCA & vertebral R SCA LSCA or IA occlusion: LIMA/RIMA ipsilateral SCA  Z]ZHZZZZ ]HC   >5  Indications for Reconstruction(Asymptomatic Severe stenotic lesions >75% Aneurysmal lesions (>4cm in RSA) EXCEPT: active Takayasu s Developmental Anolamies Esophageal or tracheal compression from abnormal RSA (usually aneurismal if symptomatic) Preparation for difficult stentgraft of the aorta [Y 2! [Y2,v t  Disease Treatment`Direct transthoracic reconstruction Remote cervical reconstruction Endovascular reconstruction M Direct ReconstructionComplete or partial (upper) median sternotomy Complete sternotomy required if aorta particularly calcified or if L SCA involved Partial sternotomy better tolerated>#  H 8Direct Reconstruction Endarterectomy best in isolated middle or distal IA lesions often lesions are continuous with aortic arch atherosclerosis and involve the ostea of other arteries off the arch challenging endarterectomy resulting thin walled artery possible flap dissection ZSR 9Direct ReconstructionBypass of IA lesion to proximal ascending aorta (borrowed from Mattox et al.) Less technically demanding than edarterectomy Partial occlusion clamp used (Lemole-Strong Clamp) 9mm-10mm graft used For multiple limbs, tailor them, handsewn*P0>o  F: Direct ReconstructionMProximal left subclavian lesions are challenging Very posterior structure (possibly easier through thoracotomy) Ligate and divide innominate vein to provide exposure via sternotomy Aberrant subclavian vessels (stenosis or aneurysm) can be resected and replaced via transposition to the common carotid and ligation of the origin ~11 >c 4 ;!Direct ReconstructionTransthoracic repair is safe and durable 0-14% mortality 94% 5 year and 88% 10 year graft patency 80% stroke-free at 10 years :)V)V s Cervical ReconstructionRecommended approach for Single lesions of the common carotid or subclavian Patients with contraindications to transthoracic approach (even multiple vessel disease) Patients with contraindication to endovascular approach B p b?"Cervical ReconstructionShort, transverse cervical incision above the clavicle Dissection between two heads of SCM Subclavian transposed to carotid Carotid transposed to subclavian  Z@#Cervical ReconstructionMust preserve vertebral and IMA Alternative: carotid-subclavian bypass Alternative: extra-anatomic bypass, retropharyngeal Use prosthetic for best longterm results  A$Cervical ReconstructionAll manner of cervical reconstruction patients Relief of symptoms ~ 100% Long term patency (10 years) 82% Stroke-free survival (10 years) 84% :/_/_Endovascular RepairAccess: Brachial artery femoral artery Cutdown during CEA Prevent embolization: retrograde approach with surgical control of neck vessels. Do not treat arteritis. Atherosclerosis and inflammatory lesions are acceptable. BZ4ZZ4,( 1Endovascular RepairStents: conflicting evidence, commonly used in SCA do not use in occluded vessels (high early re-thrombosis rate) do not use in post-vertebral SCA, thoracic outlet will crush metal framework 6  B%Endovascular RepairPrimary success rate 73-100% Complication rate 0-10% Nearly no mortality Long term durability likely inferior to surgical treatment Recurrence rate, clinically significant 16% in 4 years Summary A variety of approaches exist for the reconstruction of lesions of the supra-aortic trunks. Choice of technique and approach is based on: Patient characteristics (life expectancy, cardiac reserve, etc.) Lesion number and location Surgeon preference and experience *History: treatmentSyphilitic aneurysms of SCA 1950s endarterectomy for atherosclerotic disease of IA 1960s SCA-CA bypass, SCA-CA transposition 1994 extra-anatomic revascularization 1980s angioplasty 1990-2000s stenting/stentgraftingP  HistoryEarly 1800, first description Early 1900, Takayasu s arteritis 1960s subclavian steal syndrome described mZm/ $% & ' ( + ,-./07<=>CDEFGP   0` ̙33` ` ff3333f` 333MMM` f` f` 3>?" dd@,|?" dd@   " @ ` n?" dd@   @@``PR    @ ` ` p>> .&@ (    6Ӓ "`  T Click to edit Master title style! !$  0֒ "  RClick to edit Master text styles Second level Third level Fourth level Fifth level!     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B <xH&  VAberrant Right Subclavian 0 I S L   H  0޽h ? ___PPT10.>%/+M6Db' = @B D' = @BA?%,( < +O%,( < +DT' =%(%(D' =%(D' =4@BBBB%(D' =1:Bvisible*o3>+B#style.visibility<*G%(+  0 00(  0x 0 c $`   x 0 c $`  H 0 0޽h ? ___PPT10i.7%PC+D=' = @B +  0 40(  4x 4 c $"`   x 4 c $#U  H 4 0޽h ? ___PPT10i.7%ph+D=' = @B +   0 0(  x  c $L3`   x  c $$4  H  0޽h ? ___PPT10i.H%X+D=' = @B +   0 80(  8x 8 c $G`   x 8 c $H  H 8 0޽h ? ___PPT10i.7%)r+D=' = @B +   0 *(  x  c $V`   r  S W  H  0޽h ? ___PPT10i.N%@5+D=' = @B +   0 *(  x  c $h`   r  S iPp   H  0޽h ? ___PPT10i.N%e5+D=' = @B +   0 P0(  x  c $(`   x  c $p  H  0޽h ? ___PPT10i.\%0/+D=' = @B +  0 p0(  x  c $``   x  c $8  H  0޽h ? ___PPT10i.\%`+D=' = @B +  0 <0(  <x < c $`   x < c $<  H < 0޽h ? ___PPT10i.7%pNx+D=' = @B +  0 @0(  @x @ c $`   x @ c $  H @ 0޽h ? ___PPT10i.7%j{+D=' = @B +  0 D0(  Dx D c $`   x D c $`  H D 0޽h ? ___PPT10i.7%<€+D=' = @B +0  0  H0(  Hx H c $t`   x H c $L  H H 0޽h ? 80___PPT10.7%‰0  0 0L0(  Lx L c $`   x L c $  H L 0޽h ? 80___PPT10.7%80  0 @P0(  Px P c $`   x P c $  H P 0޽h ? 80___PPT10.7%t$  0 @$(  r  S `   r  S   H  0޽h ? 80___PPT10.k%/  0 P~(  r  S `   r  S l`   R  C *Af066004 LH  0޽h ? ___PPT10i.l%xb+D=' = @B +$  0  $(   r   S `   r   S   H   0޽h ? 80___PPT10.n%p]$  0 ($(  (r ( S  `    r ( S     H ( 0޽h ? 80___PPT10.o%\   0 PTF(  Tx T c $ `     T c $    "p`PpH T 0޽h ? ___PPT10i.7%(ǚ+D=' = @B +$  0 ,$(  ,r , S ' `    r , S h( P    H , 0޽h ? 80___PPT10.p%P B:$  0 4$(  4r 4 S <8 `    r 4 S 9    H 4 0޽h ? 80___PPT10.v%P`v$  0 <$(  <r < S XF `    r < S 0G p   H < 0޽h ? 80___PPT10.w%PEy(0  0 `X0(  Xx X c $dL `    x X c $(M 0   H X 0޽h ? 80___PPT10.7%Р$  0 $(  r  S ,Y `    r  S Z    H  0޽h ? 80___PPT10.f%p\$  0  H$(  Hr H S a `    r H S b    H H 0޽h ? 80___PPT10.y%`>0  0 p\0(  \x \ c $j `    x \ c $Xk    H \ 0޽h ? 80___PPT10.7%c  0  *(  x  c $x `    r  S | P0   H  0޽h ? ___PPT10i.;%K+D=' = @B +  0  *(   x  c $$ `    r  S      H  0޽h ? ___PPT10i.5%1+D=' = @B + 0 rj|(  |X | C pP    j | S 0 p@0    atherosclerotic oculsive disease of the vessels supplying the head and arms is found in nearly 1/3 of all patients undergoing aortography, though only a small fraction of these lesions are in the supra aortic trunks. These lesions are of interest because they affect precious territory, including the cerebral hemispheres, occular and vertebrobasilar territories, the upper extremities and cardiac vessels. Pf :H | 0޽h ? 3380___PPT10.9%7  0 (  X  C pP     S p@0    H  0޽h ? 3380___PPT10.9%@ 0 RJ(  X  C pP    J  S  p@0    TThe first description of supra aortic trunck occlusive signs and symptoms was in the early 1800, followed in the early 1900 by the description of an inflammatory process leading to  pulseless disease now known as takayasu s arteritis. In the 1960s, th anatomy of subclavian steal was described. t&    H  0޽h ? 3380___PPT10.:%[,  0 0<(  X  C pP      S \ p@0    >Surgery for supraaortic occlusive disease began as treatment for syphilitic aneurysms of the subclavian artery, initially by ligation o the innominate with poor results, eventually by resection of the aneurysms and replacement bypass grafting 75 years later, in 1965. In the 50s, endarterectomy for atherosclerotic disease of the innominate artery was first successfully performed and shortly therafter in the 1960s, subclavian to carotid bypasss and subclavian  carotid artery transposition was perfrmed. More recently, extra-anatomic, retropharyngeal bypass has been described and endovascular techniques, including angioplasty, stenting of lesions, and stent-grafting of traumatic pseudoaneurysms has been performed. These procedures will be further reviewed as we progress.  F  $ 5    $QH  0޽h ? 3380___PPT10.<%F$ 0 LD (  X  C pP    D   S | p@0     The definition of the supraaortic trunks includes the innominate, subclavian and carotids before the bifurcation. Anomalies include only one great vessel or as many as 6 trunks coming off the transverse aorta. The most common anomaly is the bovine arch where there is one trunk for the innominate and left carotid. In 6% of people, the left vertebral artery arises from between the left common carotid and left subclavian artery. Also, the right subclavian can take off the aorta separately and the carotids can come off a common trunk called the truncus bicarotidus or, very rarely, the subclavians can come off a common trunk. The relationship of the subclavian to the esophagus and the trachea can also be anomalous, and, particularly with the right subclavian, if compressing the esophagus can cause dysphasia lusoria or if compressing the trachea, causes a chronic cough. Of note, in these cases the right reccurent laryngeal nerve is not recurrent at all and takes a direct course to the trachea, laying right on the common carotid artery. Other fascinating anatomic anomalies include a right sided aortic arch which is usually found in conjunction with other cardiac anomalies but rarely can be found alone. In this radiograph I found on the internet, there is a right sided aortic arch and an abberent left subclavian artery. There is also an aneurysm of the aorta known as Kommerell s diverticulum which is a congenital dilation of the wall of the aorta :    t  [  8 Z 3Z  :  :H  0޽h ? 3380___PPT10.@%Lrr 0 (  X  C pP   "  S "p@0  " Onto diseases of the supra-aortic trunks. The most common diseases are atherosclerotic which cause an equal number of occlusive and embolic symptoms. Occlusive lesions from inflammation due to takayau s aryteritis or those due to radiation are far less common. The carotids and subclavians are also subject to dissection and aneurysmal dilation. The more distal subclavians can be affected by thoracic outlet syndrome with permanent focal changes. Congenital abnormalities account for less than 20% of lesions. b  C $  H  0޽h ? 3380___PPT10.E%0 0 \T(  X  C pP   "T  S  )"p@0  " Atherosclerotic diseases of the aortic trunk generally occur at a younger age than atherosclerotic diseases of other body regions, particularly in those with single vessel supra aortic trunk disease. There may be a more even distribution between the sexes than in other body regions. However, the risk factors are similar, including smoking, hypertension and diabetes, and not unexpectedly, concomitant coronary artery disease is common. Radiation performed for the treatment of cancers, such as breast and hodgkin s, accelerates the formation of atherosclerotic lesions in a dose dependent fashion.  UH  0޽h ? 3380___PPT10.F%@U 0 @8(  X  C pP   "8  S  p@0  " xSevere atherosclerotic disease of the supra-aortic trunks is defined as a 75% stenosis, a deep ulcerated plaque or thrombus in the lumen. Based on a 282 patient series, 40% of cases had multivessel disease and these are often due to spill over of aortic arch disease into the vessel ostea. Of those that were single vessel disease, it was most commonly the left subclavian. >Ne  H  0޽h ? 3380___PPT10.I%@U| 0 e] (  X  C pP   "]  S $>"p@0  " UIf there is multivessel disease, patients experience vertebrobasilar ischemia from low flow. If it is single vessel disease, the symptoms are generally embolic in origin. Steal syndrome is characteristically from the vertbral to the subclavian in proximal subclavian disease, and from the carotid to the subclavian in innominate disease.    &   H  0޽h ? 3380___PPT10.R%`RX 0 0h(  X  C pP   "  S HL"p@0  " jAneurysms of the supra aortic trunks can cause present because of thrombosis, embolus, compression of surrounding structures or from rupture. They were historically described in the setting of syphillis, but now this is very rare, and the majority are associated with aneurysmal dilatation of the aorta, either thoracoabdminal or ascending aorta and the ostea of the vessels are usually involved, making them harder to repair surgically. Less commonly, they are associated with takayasu s arteritis. b B !  H  0޽h ? 3380___PPT10.S%6V 0 @f(  X  C pP   "  S ,\"p@0  " hSubclavian artery aneurysms are far more common, accounting for about 2% of all peripheral aneurysms. Usually it is the middle or distal 1/3 of the vessel. Those that are extrathoracic are usually distal to the takeoff of the vertebral and are related to repetetive trauma from thoracic outlet syndrome and post-stenotic dilation of the vessel. The more proximal, intrathoracic aneurysms are related to atherosclerosis but can also be due to infection, cystic medial necrosis and marfan s [cycstic medial necrosis is focal degeneration of the elastic tissue and the muscle in the media, with the presence of mucoid material in the media. It is more frequent after 40 years of age and is twice as common in males than in females.]  G 0- gotH  0޽h ? 3380___PPT10.T%`5k" 0 `2(  X  C pP   "  S da"p@0  " 4Traumatic pseudoaneurysms or short segment dissections can be caused by deceleration injury and by penetrating iatrogenic injury secondary to misadventures during attempted placement of a central venous catheter in the subclavian vein. ,  H  0޽h ? 3380___PPT10.\% } 0 (  X  C pP   "  S y"p@0  " Other interesting aneurysmal disease includes the kommerell s diverticulum which is basically a focal, congenital aneurysmal dilation of the aorta where an anomalous right subclavian artery comes off. Takayasu s arteritis is an inflammatory condition affecting women more likely than men and most commonly in the second and third decades of life. Initially, the lesions are hypertrophic and occlusive, issues are not embolic -- symptoms are due to low flow. Later in the disease, the affected areas can become aneurysmal and then emboli are possible. Takayasu s can be compared to giant cell arteritis which affects an older population and more commonly affects the distal subclavian than the supra aortic trunks.    ( 0    }   H  H  0޽h ? 3380___PPT10.^%V/ 0 x(  X  C pP   "x  S "p@0  " Diagnosis of the described lesions begins with physical examination, including cuff pressures, palpation of pulses and listening for bruits. Some centers perform duplex of the arch but because of the air and bone in the vicinity, this is challenging and not commonly used as a screening tool. Angiography with late view to look for steal filling vessels retrograde is still the standard. Gadollinium enhanced MRI and CT Angio are now becoming more used. , H  0޽h ? 3380___PPT10.`%#G& 0 6(  X  C pP   "  S "p@0  " 8Once you ve diagnosed any one of the aforementioned lesions, Other imaging and studies that may be useful before deciding to operate include a transesophageal echocardiogram to evaluate the extent of calcification of the aorta and rule out an embolic source other than the supra-aortic trunk issues. A CT of the head to ensure that there are no recent infacts that would be negatively affected by reperfusion after intervention and finally a coronary angiogram as the concomitant CAD is as high as 40%. ,H  0޽h ? 3380___PPT10.a%p4  0 qi(  X  C pP   "i  S x"p@0  " Indications for revascularization of occlusive supra aortic trunk lesions include embolic or low-flow related transient ischemic attacks. Also, upper extremity ischemia from occlusive disease or emboli. Steal syndrome occurs when the first portion of the subclavianis occluded and the distal pressure drops below that of the vertebrobasilar junction, reversing the direction of flow, siphoning blood from the brain to the left arm! This can also occur when the IA is occluded and the common carotid and vertebral artery flow is reversed to supply the right arm. Coronary steal can also occur and result in myocardial ischemia when there is IMA CABG and there is disease of the IA or the SCA. , :gH  0޽h ? 3380___PPT10.b%P^   0 > 6 (  X  C pP   "6  S "p@0  " In order to prevent the development of complications, severe asymptomatic lesions, greater than 75% stenosis, and any incidentally found aneurysmal lesion should be repaired. The exception is active takayasu s arteritis in which case no surgery should be preformed in the asymptomatic patient. Elevated ESR and constitutional symptoms are suggestive and steroid therapy helps. An abberrant right subclavian artery can become symptomatic with dysphagia lusoria or chronic cough if it becomes aneurysmal and compresses the esophagus or trachea. If it is <4cm in diameter, observe it with serial imaging. Finally, I was surprised to read that the subclavial artery or common carotid will be transposed to preserve vertebral and left uper extremity flow while extending the proximal neck for deployment of an endograft for endovascular repair of a thoracic aortic aneurysm. d 4    $   LG 9H  0޽h ? 3380___PPT10.c%al~r  0 0 (   X   C pP   "   S "p@0  " The direct, transthoracic approach is perfommed through a median sternotomy or parital, upper median sternotomy. The partial sternotomy is better tolerated from a respiratory and post-op pain standpoint but does not give adequate access for L subclavian arterial repair or in the patient with a very calcified aorta where you re looking for a soft spot to cross clamp.      l uH   0޽h ? 3380___PPT10.j%0" 0 bZ`(  X  C pP   "Z  S "p@0  " Endarterectomy for atherosclerotic disease is best reserved for isolated middle or distal innominate lesions. Unfortunately many lesions are proximal and continuous with the arch plaque, interdigitating into the ostea of other vessels off the arch, creating a very challenging situation for endarterectomy and leaving a thin wall and edges of plaque that can serve as a lead point for a dissection. HL XY_H  0޽h ? 3380___PPT10.l%u 0 p((  X  C pP   "  S l#p@0  " *Now this borrowed image is from a trauma text, for lack of a good vascular image I could pilfer, so what is depicted here is oversewing of the origin of the IA and replacement with a graft anterior to the brachiocephalic vein. More commonly a bypass is performed as it is more feasible than endarteectomy. A partial occlusion clamp can be used nad a 9-10mm graft is required. If multiple limbs are used, they recommend tailoring them, handsewn, rather than trying to use commercial grafts. Once the reconstruciton is done, they recommend flipping the thymus between the graft and sternum. (T-berg when clamp removed to prevent embolizing air) } FG )X8u H  0޽h ? 3380___PPT10.m%lU  0 $e(  $X $ C pP   # $ S $"p@0  # gThe authors note that the anterior approach to the left subclavian vein can be challenging and that it may require the division of the innominate vein to provide exposure. Alternatively a cervical approach or posterolateral thoracotomy can be useful. P8 E @ H $ 0޽h ? 3380___PPT10.n%pJ" 0 0(  0X 0 C pP   " 0 S #p@0  " For a cervical reconstruction the incision is made just above the clavicle and the dissection is carried out between the two heads of the SCM. The omohyoid muscle is divided and the jugular and vagus are reflected laterally. The common carotid is mobilized circumfrentially and reflected medially. On the left the thoracic duct will be identified, ligated and divided. On the right unnamed lymphatic channels will be ligated and divided. Vertebral vein is tied off and divided and the subclavian artery can then be controlled, taking care not to injure the vertebral artery which comes off posteriorly. After systemic heparinization, the artery of interest is transected and re-implanted. This can be subclavian to carotid or vice versa. ':L>= _ F H 0 0޽h ? 3380___PPT10.u%(S# 0 B:8(  8X 8 C pP   #: 8 S %#p@0  # In the event that transposition is not technically feasible, bypass between the same vessels can be performed. Bypass is generlly performed more distal along the subclavian in a retro-scaleneposition. Finally, if the only target vessel is on the contralateral side, a retropharyngeal extra-anatomic bypass can be performed. The long term patency rates for the extra-anatomic bypases are not high, therefor they must be avoided when possible. In all of the above surgeries, prostetic patency is far better than vein graft. y!  1 OE "H 8 0޽h ? 3380___PPT10.v%  0 yq@ (  @X @ C pP   #q @ S 8#p@0  # The new wave of endovascular therapy has not spared the branches of the aortic arch. For atherosclerotic or inflammatory arteriopathies, angioplasty can be performed. Access can be from brachial or femoral percutaneous access or during a cutdown on the carotid for endarterectomy. 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    On-screen Show UCSF/SFIC  $ArialTimes New Roman WingdingsACS INSTRUCTOR COURSE SLIDES!Microsoft Photo Editor 3.0 PhotoSBrachiocephalic Vessel Reconstruction: Chapter 91, Rutherfords Vascular Surgery BackgroundAnatomy%Diseases of the Supra-Aortic Trunks4Atherosclerotic Disease of the Supra-Aortic Trunks4Atherosclerotic Disease of the Supra-Aortic Trunks!Symptoms: SAT occlusive diseaseSAT AneurysmsSAT AneurysmsTraumatic Aneurysms Aneurysms Diagnosis DiagnosisIndications for ReconstructionIndications for ReconstructionDisease TreatmentDirect ReconstructionDirect ReconstructionDirect ReconstructionDirect ReconstructionDirect ReconstructionCervical ReconstructionCervical ReconstructionCervical ReconstructionCervical ReconstructionEndovascular RepairEndovascular RepairEndovascular RepairSummaryHistory: treatmentHistory  Fonts UsedDesign TemplateEmbedded OLE Servers Slide Titles(_0Jason T. Lee, MDJason T. Lee, MD  !"#$%&'()*+,-./0123456789:;<=>?@ABCDEFGHIJKLMNOPQRSTUVWXYZ[\]^_`abcdefghijklmnopqrstuvwxyz{|}~      !"#$%&'()*+,-./0123456789:;<=>?@ABCDEFGHIJKLMNOPQRSTUVWXYZ[\]^_`abcdefghijklmnopqrstuvwxyz{|}~Root EntrydO)PicturesCurrent UserSummaryInformation(`UPowerPoint Document(DocumentSummaryInformation8